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Ginger supplementation supports liver health during aging through regulation of lysosomal function and molecular markers Cover

Ginger supplementation supports liver health during aging through regulation of lysosomal function and molecular markers

Open Access
|Jan 2026

Abstract

Aging is marked by hepatic structural deterioration, inflammation, and impaired autophagy. This study evaluated the effects of dietary ginger (Zingiber officinale) on liver aging in Swiss Webster mice across three age groups (3, 6, and 12 months) supplemented with 0.6% or 1.8% ginger powder for three months. Liver samples were assessed for lysosomal enzyme activity, expression of PI3K and TNF-α, histology, and ultrastructure of hepatocytes.

Aging induced hepatocellular hypertrophy, mitochondrial damage, and increased lysosomal enzyme activity, particularly arginyl aminopeptidase and acid phosphatase, and β-galactosidase - a widely recognized marker of aging, particularly cellular senescence. Ginger supplementation preserved liver morphology in young and middle-aged mice, while high doses showed mild subcellular stress in older mice. Ginger also suppressed age-related PI3K upregulation and influenced TNF-α expression, indicating potential anti-inflammatory effects.

These findings suggest that ginger modulates hepatic aging through lysosomal, inflammatory, and metabolic pathways in an age- and dose-dependent manner. Moderate intake appears most beneficial, supporting its potential as a nutraceutical for liver health during aging.

Language: English
Page range: 385 - 404
Accepted on: Sep 30, 2025
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Published on: Jan 12, 2026
In partnership with: Paradigm Publishing Services
Publication frequency: 4 issues per year

© 2026 Maima Matin, Małgorzata Łysek-Gładysinska, Natalia Ksepka, Sara Frazzini, Anna Wieczorek, Artur Józwik, published by Institute of Genetics and Animal Biotechnology of the Polish Academy of Sciences
This work is licensed under the Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 License.