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Management of Pulmonary Hypertension in Left Heart Disease Cover

Management of Pulmonary Hypertension in Left Heart Disease

Open Access
|Jul 2021

Figures & Tables

Table 1

Clinical and pathobiological characteristics of pulmonary arterial hypertension (PAH) and pulmonary hypertension (PH) due to left heart diseases. ACE: angiotensin converting enzyme; ARB: angiotensin receptor antagonist; ARNI: angiotensin receptor neprilysin inhibitor; ERA: endothelin receptor antagonist; F: female; HF: heart failure; HFpEF: heart failure with preserved ejection fraction; HFrEF: heart failure with reduced ejection fraction; LA: left atrium; LBBB: left bundle branch block; LHF: left heart failure; LV: left ventricle; M: male; MRA: mineral-corticoid receptor antagonist; PAWP: pulmonary artery wedge pressure; PDE5: phosphodiesterase-5; PVOD: pulmonary veno-occlusive disease; RA: right atrium; RBBB: right bundle branch block; RHF: right heart failure; RV: right ventricle; sGC: soluble guanylate cyclase; SGLT2: sodium-glucose cotransporter 2; TR: tricuspid regurgitation; VCO2: CO2 output; VE: ventilation; VO2: oxygen uptake.

GROUP 1 PH (PAH)GROUP 2 PH (PH-LHD)
Pathobiology
  • Hemodynamics

  • Histopathology

  • Precapillary (PAWP < 15 mm Hg)

  • Dysregulation of proliferative (↑) and apoptotic (↓) signals

  • Distal pulmonary arteries: intima-medial hypertrophy; inflammation → endothelial-mesenchymal transition → muscularization; abnormal vasoconstrictive response

    Veins: in PVOD, massive fibrous intima thickening with occlusion of small preseptal venules

  • Postcapillary (PAWP ≥ 15 mm Hg)

  • Capillaries: (early) ↑ endothelial permeability (collagen fragmentation), and ↓ alveolar fluid clearance (↓ Na-K ATP-ase function); (late) thickening extracellular matrix Arterioles: (early) vasoconstriction → (late) muscularization (intima-medial hypertrophy/hyperplasia)

    Veins: (unclear) “arterialization”

Phenotype/demographic
  • Age

  • Sex

  • CV risk factors

  • Younger

  • F:M ͠ 2:1

  • +

  • Older (> in HFpEF)

  • M > F in HFrEF; F > M in HFpEF

  • +++

Clinical
  • Symptoms

  • ECG

  • Echocardiography

  • Exercise


  • Syncope; RHF (visceral congestion, hepatomegaly, lower limb edema)

  • Sinus tachycardia; pulmonary P wave, right axis deviation, RBBB, neg T-wave V1-V3

  • RV/LV ratio > 1, D-shape of LV, RA enlargement > LA, significant TR, pericardial effusion

  • Slope VE/VCO2 increase +++, exercise-induced O2-desaturation (+/–)


  • LHF (orthopnea, exercise-induced dyspnea, systemic hypotension)

  • Atrial fib (frequent); negative P-wave V1-V2, LV hypertrophy, left axis deviation, LBBB

  • Normal RV/LV ratio, LV dilation/ dysfunction and/or hypertrophy, high E/E’ ratio, LA enlargement

  • Early AT (< 40% predicted VO2), slope VE/VCO2 increase –/+, oscillatory exercise breathing

Therapy
  • Targeting the heart: none

  • Targeting the pulmonary circulation:

    • ERA (bosentan, ambrisentan, macitentan)

    • PDE5 inhibitors (sildenafil, tadalafil)

    • sGC stimulators (riociguat)

    • Prostacyclin analogues or agonists (epoprostenol, iloprost, treprostinil, selexipag)

  • Targeting the heart:

    • Beta-blockers (for HFrEF)

    • ACE-inhibitors/ARB (for HFrEF)

    • ARNI (for HFrEF)

    • MRA (for HFrEF)

    • SGLT2 inhibitors (for diabetic patients)

    • Encouraging results from vericiguat (HFrEF)

  • Targeting the pulmonary circulation: No established/recommended treatment

Table 2

Design and main results of randomized clinical trials exploring pulmonary arterial hypertension (PAH)-specific drugs in pulmonary hypertension (PH) due to left heart diseases.33,34,35,36,37,38,39,40,41,42 6MWD: 6-minute walk distance; BNP: B-type natriuretic peptide; CI: cardiac index; CO: cardiac output; CPET: cardiopulmonary exercise test; DPG: diastolic pulmonary gradient; EOB: exercise oscillatory breathing; HF: heart failure; HFpEF: heart failure with preserved ejection fraction; HFrEF: heart failure with reduced ejection fraction; LV: left ventricle; mPAP: mean pulmonary artery pressure; NYHA: New York Heart Association (functional class); PAWP: pulmonary artery wedge pressure; PVR: pulmonary vascular resistance; QoL: quality of life; RAP: right atrial pressure; RHC: right heart catheterization; RV: right ventricle; sPAP: systolic pulmonary artery pressure; VO2: oxygen uptake; WU: Wood units

STUDYTARGET PATIENTSDRUG (N)END POINTSRESULTS
FIRST33Severe HFrEF (LVEF < 25%)
NYHA IIIB/IV
Congestive HF (PAWP > 15, CI < 2.2)
Epoprostenol (237) vs conventional medical therapy (234)Primary: death; major event (death, need for mechanical ventilation, inotropic drugs, mechanical circulatory support)
Secondary: 6MWD, QoL, clinical status at 3 months
Early termination due to increased mortality for HF in treatment group
Lewis GD et al.34HFrEF (LVEF < 40%)
NYHA II-IV
PH (mPAP > 25 mm Hg at RHC)
Sildenafil (17) vs placebo (17), for 12 weeksPrimary: VO2 peak
Secondary: 6MWD, PVR
Increase peak VO2, improved 6MWD, decrease PVR in treatment group
Guazzi M, et al.35HFpEF (LVEF > 50%)
NYHA II-IV
PH (sPAP > 40 mm Hg at echo)
Sildenafil (22) vs placebo (22), for 1 yearPrimary: pulmonary hemodynamics; RV function (TAPSE)
Secondary: QoL
Significant reduction in RAP, mPAP, PAWP and PVR; improvement in RV function, CI and QoL
Guazzi M, et al.36HFrEF (LVEF < 45%)
PH (mPAP 25–35 mm Hg at RHC)
EOB at CPET
Sildenafil (16) vs placebo (16) for 1 yearRespiratory pattern during CPET
Pulmonary hemodynamics
Significant EOB reversal in treatment group
Significant reduction in pulmonary pressure and PVR, and increase in CO in treatment group
Hoendermis ES, et al.37HFpEF (LVEF ≥ 45%)
PH (mPAP > 25, PAWP > 15 mm Hg)
Sildenafil (26) vs placebo (26), for 12 weeksChange in mPAP, PAWP, CO and peak VO2No significant differences
Liu LC, et al.38HFpEF (LVEF ≥ 45%)
NYHA II-IV
PH (mPAP > 25, PAWP > 15 mm Hg)
Sildenafil (26) vs placebo (26), for 12 weeksEchocardiographic parameters (RV/LV dimensions and function)
CPET, QoL
No significant differences
SIOVAC39PH (mPAP > 30 mm Hg at RHC)
Left-side valvular replacement or repair 1 year before
Sildenafil (104) vs placebo (96), for 6 monthsPrimary: composite clinical score (death or HF + NYHA class + QoL)
Secondary: clinical score components, 6MWD, BNP, echocardiography
Significant worsening in clinical status of patients in sildenafil group (driven by higher risk of readmission for HF).
No differences in sPAP, 6MWD, NYHA class
BADDHY40HFpEF (LVEF ≥ 50%)
PH (mPAP > 25 mm Hg, PAWP > 15 mm Hg at RHC)
RV dysfunction (echo)
Bosentan (9) vs placebo (11) for 12 weeks6MWD
sPAP and RAP estimated by echocardiography
Insignificant trend in increase of 6MWD in placebo group Acute HF event in 3 patients in bosentan group vs 1 patient in placebo group
LEPHT41HFrEF (LVEF ≤ 40%)
NYHA II-IV
PH (mPAP ≥ 25 mm Hg at RHC)
Riociguat (132) vs placebo (69), for 16 weeksPrimary: mPAP changes
Secondary: hemodynamic parameters
Exploratory: clinical worsening, death, HF hospitalization, 6MWD, NYHA class, QoL
No significant changes in mPAP
Significant increase in CI and decrease in PVR in riociguat group.
MELODY-142HFpEF and HFrEF (LVEF > 35%)
NYHA II-IV
CpcPH (mPAP ≥ 25, PAWP > 15, DPG ≥ 7, PVR > 3.0 WU)
Macitentan (31) vs placebo (32) for 12 weeksPrimary: safety and tolerability (fluid retention, worsening NYHA class)
Exploratory: changes in hemodynamics, NT-proBNP, 6MWD
More patients in macitentan group than in placebo group experienced fluid retention
No significant differences in any of the exploratory endpoints
ACEAngiotensin converting enzyme
ARBAngiotensin receptor antagonist
ARNIAngiotensin receptor neprilysin inhibitor
ATAnaerobic threshold
CICardiac index
COCardiac output
CpcPHCombined post- and precapillary pulmonary hypertension
CPETCardiopulmonary exercise test
DPGDiastolic pulmonary gradient
EOBExercise oscillatory breathing
ERAEndothelin receptor antagonist
iPAHIdiopathic pulmonary arterial hypertension
IpcPHIsolated postcapillary pulmonary hypertension
HFHeart failure
HFpEFHeart failure with preserved ejection fraction
HFrEFHeart failure with reduced ejection fraction
LALeft atrium
LAPLeft atrium pressure
LBBBLeft bundle branch block
LHDLeft heart disease
LVLeft ventricle
LVADLeft ventricle assistance device
mPAPMean pulmonary artery pressure
MRAMineral-corticoid receptor antagonist
NONitric oxide
NT-proBNPN-terminal pro–brain natriuretic peptide
NYHANew York Heart Association (functional class)
PAPPulmonary artery pressure
PAWPPulmonary artery wedge pressure
PDE-5Phosphodiesterase-5
PHPulmonary hypertension
PH-LHDPulmonary hypertension due to left heart disease
PVODPulmonary veno-occlusive disease
PVRPulmonary vascular resistance
QoLQuality of life
RARight atrium
RBBBRight bundle branch block
RHFRight heart failure
RVRight ventricle
sGCSoluble guanylate cyclase
SGLT2Sodium-glucose cotransporter 2
TRTricuspid regurgitation
VCO2CO2 output
VHDValvular heart disease
VO2Oxygen uptake
VEVentilation
6MWD6-Minute walking distance
DOI: https://doi.org/10.14797/RKQN5397 | Journal eISSN: 1947-6108
Language: English
Page range: 115 - 123
Accepted on: Nov 18, 2020
Published on: Jul 1, 2021
Published by: Houston Methodist DeBakey Heart & Vascular Center
In partnership with: Paradigm Publishing Services

© 2021 Francesca Macera, Jean-Luc Vachiéry, published by Houston Methodist DeBakey Heart & Vascular Center
This work is licensed under the Creative Commons Attribution-NonCommercial 4.0 License.