Figure 1
Incidentally perihepatic lesion in a 49‑year‑old man on contrast‑enhanced CT imaging. Axial (A) and sagittal images (C) showed a well‑defined enhancing mass that slightly deformed the diaphragmatic surface of the liver (white arrows) into a crescent shape (negative “embedded organ sign, white arrow heads) with dull edge (negative “beak sign”). Axial image (B) showed nodules into the left subdiaphragmatic space having the same density as the right peri hepatic lesion (*).
Figure 2
MRI images of the upper abdomen. Axial fat‑suppressed T2‑weighted image (A) showed that the right subdiaphragmatic lesion is slightly hyperintense (arrow). Axial T1‑in phase GRE image (B) and axial T1‑out‑of‑phase GRE image (C) showed that the lesion is slightly hypointense with no signal drop (arrow). Subdiaphragmatic fat is responsible of a fat chemical shift artifact between the lesion and the liver (arrowheads). Axial post‑contrast T1 mDixon MRI (D, E, F) showed progressive enhancement of the lesion (arrows), similar to the left subdiaphgramatic nodule (*).
Figure 3
Axial Tc‑99m ‑ RBC scintigraphy (A, B) and fused with axial portal‑venous CT images (C, D) showed an increased homogenous radionuclide uptake in the perihepatic lesion (arrows) and in the left subdiaphragmatic mass corresponding to splenosis (curved arrows).