References
- Adonaylo VN, Otieza PI. (1999). Pb2+ promotes lipid peroxidation and alteration in membrane physical properties. Toxicology 132: 19–32.
- Akitane M, Jiankang L, Xiaryan W, Motoko, K. (1994). Free Radical Scavenging by Brain Homogenate: Implication to free Radical damage and antioxidant defence in brain; Neurochem. Int 24: 201–207.
- Al-Attar AM. (2011). Antioxidant effect of vitamin E treatment on some heavy metals-induced renal and testicular injuries in male mice. Saudi J Biol Sci 18(1): 63–72.
- Armstrong JS. (2006). The role of the mitochondrial permeability transition in cell death. Mitochondrion 6(5): 225–34.
- ATSDR (Agency for Toxic Substances and Disease Registry), 2007. Public Health Statement, Lead, U.S. Department of Health and Human Services, Public Health Services.
- Basha CD, Reddy RG. (2015). Long term changes in brain cholinergic system and behavior in rats following gestationalexposure to lead: protective effect of calcium supplement. Interdiscip Toxicol 8(4): 159–68.
- Basha DC, Basha SS, Reddy GR. (2012). Lead-induced cardiac and hemato-logical alterations in aging Wistar male rats: alleviating effects of nutrient metal mixture. Biogerontology 13(4): 359–68.
- Battacharayya MH. (1983). Bioavailability of orally administered cadmium and lead to the mother, fetus, and neotal during pregnancy and lactation. Sci. Total Environ 28: 327–42.
- Beier EE, Sheu TJ, Dang D, Holz JD, Ubayawardena R, Babij P, Puzas, JE. (2015). Heavy metal ion regulation of gene expression: mechanisms by which lead inhibits osteoblastic bone forming activity through modulation of the Wnt/-catenin signaling pathway. J. Biol. Chem 290: 18216–18226.
- Chance B, Maehly AC. (1955). Assay of catalase and peroxidises. Meth. Enzymol I 11: 764–775.
- Basha DC, Reddy NS, Rani MU, Reddy GR. (2014). Age related changes in aminergic system and behavior following lead exposure: protection with-essential metal supplements. Neurosci Res 78: 81–9.
- Dietrich KN. (1991). Human foetal lead exposure: intrauterine grow, maturation and postnatal development. Fundam.Appl. Toxicol 16: 17–19.
- Dobrakowski M, Pawlas N, Kasperczyk A, Kozłowska A, Olewińska E, Machoń-Grecka A, Kasperczyk S. (2017). Oxidative DNA damage and oxidative stress in lead-exposed workers. Hum Exp Toxicol 36(7): 744–754.
- EFSA (European Food Safety Authority). (2012). Lead dietary exposure in the European population. EFSA J. 10: 2831.
- Bruns FH, Bergmeyer HU. (1965). Fructose-1,6 di phosphatase aldolase. In: Methods of enzymatic analysis. Bergmeyer, H.U. Academic press, New York. 724–731.
- Flora G, Gupta D, Tiwari A. (2012). Toxicity of lead: a review with recent updates. Interdiscip. Toxicol 5: 47–58
- Flora SJ, Pande M, Mehta A. (2003). Beneficial effect of combined administration of some naturally occurring antioxidants (vitamins) and thiol chelators in the treatment of chronic lead intoxication. Chem. Biol. Interact 145: 267–280.
- Gao A, Lu XT, Li QY, Tian L. (210). Effect of the delta-aminolevulinic acid dehydratase gene polymorphism on renal andneurobehavioral function in workers exposed to lead in China. Sci Total Environ 408(19): 4052–5.
- Gardella C. (2001). Lead exposure in pregnancy: a review of the literature and argument for routine perinatal screening. Obstet Gyn Survey 56: 231–8.
- Gottipolu RR, Davuljigari CB. (2014). Perinatal exposure to lead: reduction in alterations of brain mitochondrial antioxidant systemwith calcium supplement. Biol Trace Elem Res 162(1–3): 270–7.
- Gurer H, Ercal N. (2000). Can antioxidants be beneficial in the treatment of lead poisoning? Free Radic. Biol. Med. 29: 927–945.
- Gutowicz M. (2011). The influence of reactive oxygen species on the central nervous system. Postepy Hig Med Dosw (Online) 65: 104–13.
- Lai JC, Clark JB. (1979). Preparation of synaptic and non-synaptic mitochondria from mammalian brain. Meth Enzymol 55: 51–60.
- Jarrar BM, Taib NT. (2012). Histological and histochemical alterations in the liver induced by lead chronic toxicity. Saudi J Biol Sci 19(2): 203–10.
- Jia Q, Ha X, Yang Z, Hui L, Yang X. (2012). Oxidative stress: a possible mechanism for lead-induced apoptosis and nephrotoxicity. Toxicol Mech Methods. 22(9): 705–10.
- Kelman BJ, Walter BK. (1980). Transplacental movement of inorganic lead from mother to fetus. Proc. Soc. Exp. Biol. Med 163: 278–282.
- Kilikdar D, Mukherjee D, Mitra E, Ghosh AK, Basu A, Chandra AM, Bandyoapdhyay D. (2011). Protective effect of aqueous garlic extract against lead-induced hepatic injury in rats. Indian J Exp Biol. 49(7): 498–510.
- Kluska K, Adamczyk J, Krężel A. (2018). Metal binding properties, stability and reactivity of zinc fingers Coordination. Chemistry Reviews 367: 18–64
- Kuhad, A, Chopra K. (2007). Curcumin attenuates diabetic encephalopathy in rat: Behavioral and biochemical evidence. European Journal of Pain 576: 34.
- Liu KS, Hao JH, Zeng Y, Dai FC, Gu PQ. (2013). Neurotoxicity and biomarkers of lead exposure: a review. Chin Med Sci J 28(3): 178–88.
- Lopes AC, Peixe TS, Mesas AE, Paoliello MM. (2016). Lead Exposure and Oxidative Stress: A Systematic Review. Rev Environ Contam Toxicol 236: 193–238.
- Lowry OH, Rosenbrough NJ, Farr AL, Randall RJ. (1951). Protein measurement with Folin-phenol reagent. J. Biol. Chem 193: 265–275.
- Ma L, Liu JY, Dong JX, Xiao Q, Zhao J, Jiang, FL. (2017). Toxicity of Pb2+ on rat liver mitochondria induced by oxidative stress and mitochondrial permeability transition. Toxicol. Res 6: 822–830.
- Machartová V, Racek J, Kohout J, Senft V, Trefil L. (2000). Effect of anti-oxidant therapy on indicators of free radical activity in workers at risk of leadexposure. Vnitr Lek. 46(8): 444–6.
- Misra HP, Fridovich I. (1972). The role of superoxide anion in the auto-oxidation of epinephrine and a simple assay for superoxide dismutase. J. Biol. Chem 247: 3170–3175.
- Sreenivasulu N, Kumar RM, Basha DC, Rajarami GR. (2015). Lead induced alterations in behavior and brain cholinergic system in female rats: ameliorative effect of α-tocopherol. Indo American Journal of Pharmaceutical Research 5(10): 3281–3292
- Nachlas MM, Marguil SI, Seligman, AM. (1960). A corlorimetric method for determination of succinate dehydrogenase activity. J. Biol. Chem 235: 499–505.
- Ohkawa H, Ohishi N, Yagi K. (1979). Assay for lipid peroxides in animal tissues by thiobarbituric acid reaction. Anal. Biochem 95(2): 351–358.
- Pande M, Flora SJ. (2002). Lead induced oxidative damage and its response to combined administration of alpha-lipoic acid and succimers in rats. Toxicology 15: 177(2–3): 187–96.
- Patra RC, Amiya K, Rautray, D. (2011). Oxidative Stress in Lead and Cadmium Toxicity and Its Amelioration. Vet Med Int. 2011: 457327.
- Patra RC, Swarup D, Dwivedi SK. (2001). Antioxidant effects of alpha tocopherol, ascorbic acid and l-methionine on lead induced oxidative stress to the liver kidney and brain in rats. Toxicology 162: 81–88.
- Patriarca M, Menditto, A, Rossi B, Lyon TDB, Fell GS. (2000). Enivormental exposure to metals of newborn, infants and young children. Microchem 1–67: 351–361.
- Patrick L. (2006). Lead toxicity. Part II. The role of free radicals damage and the use of antioxidants in the pathology and treatment of lead toxicity. Altern. Med. Rev. 11: 114–128.
- Prameelamma Y, Swami, KS. (1975). Glutathione dehydrogenase activity in normal and denervated gastrocnemius muscle of frog, Rana hyxadactyla. Curr. Sci 44: 739–740.
- Prasanthi RPJ, Devi CB, Basha DC, Reddy NS, Reddy GR. (2010). Calcium and zinc supplementation protects lead (Pb)-induced perturbations in anti-oxidant enzymes and lipid peroxidation in developing mouse brain. Int. J. Devl. Neurosci 28: 161–167.
- Pushpakiran G, Mahalakshmi K, Anuradha CV. (2004). Taurine restores ethanol-induced depletion of antioxidants and attenuates oxidative stress in rat tissues. Amino Acids 27: 91–9
- Ramanathan K, Shila S, Kumaran S, Paneerselvan C. (2003). Ascorbic acid and alpha-tocopherol as potent modulators on arsenic induced toxicity in mitochondria. Journal of Nutritional Biochemistry 14: 416–420.
- Rendón-Ramírez AL, Maldonado-Vega M, Quintanar-Escorza MA, Hernández G, Arévalo-Rivas BI, Zentella-Dehesa A, Calderón-Salinas JV. (2014). Effect of vitamin E and C supplementation on oxidative damage and total antioxidant capacity in lead-exposed workers. Environ Toxicol Pharmacol 37(1): 45–54.
- Rendón-Ramírez A, Cerbón-Solorzano J, Maldonado-Vega M, Quintanar-Escorza MA, Calderón-Salinas JV. (2007). Vitamin-E reduces the oxidative damage on delta-aminolevulinic dehydratase induced by lead intoxication in rat erythrocytes. Toxicol In Vitro. 21: 1121–1126.
- Rotruck JT, Pope AL, Ganther HE, Swanson AB, Hafeman DG, Hoekstra WG. (1973). Biochemical role as a component of glutathione peroxidase. Science 179: 588–590.
- Sajitha GR, Jose R, Andrews A, Ajantha KG, Augustine P, Augusti KT. (2010). Garlic Oil and Vitamin E Prevent the Adverse Effects of Lead Acetate and Ethanol Separately as well as in Combination in the Drinking Water of Rats. Indian J Clin Biochem 25(3): 280–8.
- Salehi I, Karamian R, Komaki A, Tahmasebi L, Taheri M, Nazari M, Shahidi S, Sarihi A. Effects of vitamin E on lead-induced impairments in hippocampal synaptic plasticity. Brain Res 10: 629: 270–81.
- Sandhir R, Julka D, Gill KD. (1994). Lipoperoxidative damage on lead treatment in rat brain and its implications on membrane bound enzymes. Pharmacol Toxicol 74: 66–71
- Sara EE, Hongfei G, Neal Fedarko, Amy DeZern, Linda PF, Qian-Li Xue, Sean Leng, Brock Beamer, Jeremy DW. (2008). Glutathione Peroxidase Enzyme Activity in Aging. J Gerontol A Biol Sci Med Sci 63(5): 505–509.
- Sousa CA, Soares EV. (2014). Mitochondria are the main source and one of the targets of Pb (lead)-induced oxidative stress in the yeast Saccharomyces Cerevisiae. Appl Microbiol Biotechnol 98(11): 5153–60.
- Stanton RC. (2012). Glucose-6-phosphate dehydrogenase, NADPH, and cell survival. IUBMB Life 64(5): 362–9.
- Stohs SJ, Bagchi D. (1995). Oxidative mechanisms in the toxicity of metal ions. Free Radic Biol Med 18(2): 321–363.
- Traber MG, Stevens JF. (2011). Vitamins C and E beneficial effects from a mechanistic perspective. Free Radic Biol Med 51: 1000–1013
- Viani P, Cervato G, Fiorilli A, Cestaro B. (1991). Age related differences in synaptosomal peroxidative damage and membrane Proteins. J Neurochem 56: 253–258.
- Villeda-Hernandez J, Barroso-Moguel R, Mendez-Armenta, M, Nava-Ruiz C, Huerta-Romero R, Rios C. (2001). Enhanced brain regional lipid peroxidation in developing rats exposed to low level lead acetate. Brain Res Bull 55(2): 247–51.
- Wrangler JA, Richardson JS. (1991). Oxygen Free Radicals and brain dysfunction. Int J Neurosci 57: 1–17.
- Lee YL, Lardy HA. (1965). Influence of thyroid hormones on L- glycerol phosphate dehydrogenase and other dehydrogenases in various organs of rat. J Biol Chem 240: 1427–1432.
- Zawia NH, Harry GJ. (1996). Developmental exposure to lead interferes with glial and neuronal differential gene expression in the rat cerebellum. Toxicol Appl Pharmacol 138(1): 43–7.