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Leptin alleviated ovarian ischemia-reperfusion injury in rats via modulation of Sirt-1/Nrf2 and TLR4/NF-kB/caspase-3 signaling pathways Cover

Leptin alleviated ovarian ischemia-reperfusion injury in rats via modulation of Sirt-1/Nrf2 and TLR4/NF-kB/caspase-3 signaling pathways

Endocrine Regulations
Volume 57 (2023): Issue 1 (January 2023)
By: Heba A. Abdel-Hamid,  Nehad MR. Abdel Maqsoud,  Nisreen DM. Toni,  Rasha F. Ahmed and  Elshymaa A. Abdel-Hakeem  
Open Access
|Feb 2023

Authors

Heba A. Abdel-Hamid

heba.abdelhameed@minia.edu.eg

Medical Physiology Department, Faculty of Medicine, Minia University, Minia, Egypt
Medical Physiology Department, Faculty of Medicine, Al-Baha University, KSA

Nehad MR. Abdel Maqsoud

Pathology Department, Faculty of Medicine, Delegated to Deraya University New Minia City, Minia University, Minia, Egypt

Nisreen DM. Toni

Pathology Department, Faculty of Medicine, Minia University, Minia, Egypt

Rasha F. Ahmed

Biochemistry Department, Faculty of Medicine, Minia University, Minia, Egypt

Elshymaa A. Abdel-Hakeem

Medical Physiology Department, Faculty of Medicine, Minia University, Minia, Egypt
DOI: https://doi.org/10.2478/enr-2023-0004 | Journal eISSN: 1336-0329 | Journal ISSN: 1210-0668
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Language: English
Page range: 25 - 36
Published on: Feb 8, 2023
Published by: Slovak Academy of Sciences, Institute of Experimental Endocrinology
In partnership with: Paradigm Publishing Services
Publication frequency: 1 issue per year
Keywords:
ischemia,
reperfusion,
ovary,
leptin,
sirtuin 1,
toll like receptor-4
Related subjects:
Life sciences,
Molecular biology,
Neurobiology,
Medicine,
Basic medical science,
Basic medical science, other,
Clinical medicine,
Internal medicine,
Endocrinology, diabetology

© 2023 Heba A. Abdel-Hamid, Nehad MR. Abdel Maqsoud, Nisreen DM. Toni, Rasha F. Ahmed, Elshymaa A. Abdel-Hakeem, published by Slovak Academy of Sciences, Institute of Experimental Endocrinology
This work is licensed under the Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 License.

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