Changes in erythrocyte deformability during day and possible role of melatonin
References
- Berker M, Dikmenoglu N, Bozkurt G, Ergonul Z, Ozgen T. Hemorheology, melatonin and pinealectomy. What’s the relationship? An experimental study. Clin Hemorheol Microcirc 30, 47-52, 2004
- Benitez-King G. Melatonin as a cytoskeletal modulator: implications for cell physiology and disease. J Pineal Res 40, 1-9, 2006.10.1111/j.1600-079X.2005.00282.x16313492
- Chakravarty S, Rizvi SI. Circadian modulation of sodium-potassium ATPase and sodium - proton exchanger in human erythrocytes: in vitro eff ect of melatonin. Cell Mol Bio 57, 80-86, 2011.
- Csernus V, Mess B. Biorhythms and pineal gland. Neuro Endocrinol Lett 24, 404-411, 2003.
- Dikmenoglu N, Ileri E, Seringec N, Ercil D. Melatonin prevents lipid peroxidation in human erythrocytes but augments deterioration of deformability aft er in vitro oxidative stress. Clin Hemorheol Microcirc 40, 235-242, 2008.10.3233/CH-2008-1132
- Gumustekin M, Tekmen I, Guneli E, Tugyan K, Topcu A, Ergonen AT, Ozdemir MH, Uysal N, Bediz CS. Short-term melatonin treatment improved diabetic nephropathy but did not aff ect hemorheological changes in diabetic rats. Pharmazie 62, 693-698, 2007.
- Herichova I. Changes of physiological functions induced by shift work. Endocr Regul 47, 159-170, 2013.10.4149/endo_2013_03_15923889486
- Muller JE, Ludmer PL, Willich SN, Tofl er GH, Aylmer G, Klangos I, Stone PH. Circadian variation in the frequency of sudden cardiac death. Circulation 75, 131-138, 1987.10.1161/01.CIR.75.1.131
- Radosinska J, Vrbjar N. Th e role of red blood cell deformability and Na,K-ATPase function in selected risk factors of cardiovascular diseases in humans: focus on hypertension, diabetes mellitus and hypercholesterolemia. Physiol Res 65 Suppl 1, S43−S54, 2016.10.33549/physiolres.93340227643939
- Radosinska J, Mezesova L, Okruhlicova L, Frimmel K, Breierova E, Bartekova M, Vrbjar N. Eff ect of yeast biomass with high content of carotenoids on erythrocyte deformability, NO production and Na,K-ATPase activity in healthy and LPS treated rats. Clin Hemorheol Microcirc 64, 125-134, 2016.10.3233/CH-16205127002892
- Raj K, Bhatia R, Prasad K, Srivastava MV, Vishnubhatla S, Singh MB. Seasonal diff erences and circadian variation in stroke occurrence and stroke subtypes. J Stroke Cerebrovasc Dis 24, 10-16, 2015.10.1016/j.jstrokecerebrovasdis.2014.07.05125284717
- Schmid-Schonbein H. Microrheology of erythrocytes, blood viscosity, and the distribution of blood fl ow in the microcirculation. Int Rev Physiol 9, 1-62, 1976.
- Segura D, Eblen-Zajjur A, Proverbio F, Proverbio T, Carrera F, Caruso-Neves C, Marin R. A blood plasma inhibitor is responsible for circadian changes in rat renal Na,K-ATPase activity. Int J Biochem Cell Biol 36, 2054-2065, 2004.10.1016/j.biocel.2004.03.00515203118
- Yerer MB, Aydogan S. Th e importance of circadian rhythm alterations in erythrocyte deformability. Clin Hemorheol Microcirc 35, 143-147, 2006.
Language: English
Page range: 17 - 20
Published on: Feb 16, 2018
Published by: Slovak Academy of Sciences, Mathematical Institute
In partnership with: Paradigm Publishing Services
Publication frequency: 1 times per year
Related subjects:
© 2018 Rastislav Vazan, Katarina Plauterova, Gabriela Porubska, Jana Radosinska, published by Slovak Academy of Sciences, Mathematical Institute
This work is licensed under the Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 License.