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Analysis of gene expression related to adrenal physiological functions in Huntington’s disease mouse model Cover

Analysis of gene expression related to adrenal physiological functions in Huntington’s disease mouse model

Open Access
|Dec 2025

Abstract

Huntington’s disease (HD) is a genetic disorder that arises from a mutation in the huntingtin gene, leading to the formation of toxic aggregates. These elements are predominantly deposited in neurons, with smaller amounts found in the cells of numerous peripheral organs. One such example is the adrenal glands, which are responsible for the production of catecholamines, such as dopamine, norepinephrine, and epinephrine, in addition to steroid hormones, including cortisol and aldosterone. Abnormalities in the concentrations of these produced factors have been demonstrated in both patients and animal models. However, there is a lack of studies related to the expression profile of genes responsible for adrenal physiological functions. The objective of this study was to ascertain whether there have been alterations in the expression of genes responsible for the processes of steroidogenesis and catechola-mine production in the adrenal glands of the R6/1 mouse model of HD. To this purpose, we employed quantitative polymerase chain reaction (qPCR) analyses to ascertain the relative expression levels of the genes responsible for these two processes. A significant decrease in the expression of genes involved in the catecholamine production pathway was observed, while no change in the expression of genes related to steroidogenesis was detected. This finding suggests that reduced gene expression may contribute to the impaired production of catecholamines in R6/1 mice.

Language: English
Page range: 31 - 39
Submitted on: Sep 1, 2025
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Accepted on: Oct 7, 2025
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Published on: Dec 18, 2025
In partnership with: Paradigm Publishing Services
Publication frequency: 4 issues per year

© 2025 Anna Olechnowicz, Małgorzata Blatkiewicz, Mark Isalan, Agnieszka Koc, Michal Mielcarek, Marcin Rucinski, published by Foundation for Cell Biology and Molecular Biology
This work is licensed under the Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 License.