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The influence of renin angiotensin aldosterone system blockers on asymmetric dimethylarginine levels in patients with chronic glomerulonephritis Cover

The influence of renin angiotensin aldosterone system blockers on asymmetric dimethylarginine levels in patients with chronic glomerulonephritis

Open Access
|Jan 2017

References

  1. 1. Bonetti P.O., Lerman L.O., Lerman A. Endothelial dysfunction: a marker of atherosclerotic risk. Arterioscler Thromb Vasc Biol., 23,168-175, 2003.10.1161/01.ATV.0000051384.43104.FC
  2. 2. Böger R.H. et al. Asymmetric Dimethylarginine (ADMA): A Novel Risk Factor for Endothelial Dysfunction, Its Role in Hypercholesterolemia. Circulation, 98, 1842-1847, 1998.10.1161/01.CIR.98.18.1842
  3. 3. Caglar K. et al. ADMA, proteinuria, and insulin resistance in non-diabetic stage I chronic kidney disease. Kidney Int., 70, 781-787, 2006.10.1038/sj.ki.5001632
  4. 4. Colonna De Gennaro V. et al. Asymmetric dimethylarginine (ADMA): an endogenous inhibitor of nitric oxide synthase and a novel cardiovascular risk molecule. Med Sci Monit., 15, RA91-101, 2009.
  5. 5. Deckert T., Feldt-Rasmussen B., Borch-Johnsen K. Albuminuria reflects widespread vascular damage. The Steno hypothesis. Diabetologia, 32, 219-226, 1989.10.1007/BF00285287
  6. 6. Fliser D. et al. Asymmetric dimethylarginine and progression of chronic kidney disease: the mild to moderate kidney disease study. J Am Soc Nephrol., 16, 2456-2461, 2005.10.1681/ASN.2005020179
  7. 7. Fujii H. et al. Renin-Angiotensin System Inhibitors Reduce Serum Asymmetric Dimethylarginin Levels and Oxidative Stress in Normotensive Patients with Chronic Kidney Disease. Nephron Extra, 4, 18-25, 2014.10.1159/000358886
  8. 8. Fujimi-Hayashida A. et a l. Association of asymmetric dimethylarginine with severity of kidney injury and decline in kidney function in IgA nephropathy. Am J Nephrol., 33,1-6,2011.10.1159/000322367
  9. 9. Hibbs J.B., Vavrin Z., Taintor R.R. L-Arginine is required for expression of the activated macrophage effector mechanism causing selective metabolic inhibition in target cells. J Immunol.,138: 550-565,1987.10.4049/jimmunol.138.2.550
  10. 10. Makino H. et al. INNOVATION Study Group: Prevention of transition from incipient to overt nephropathy with telmisartan in patients with type 2 diabetes. Diabetes Care, 30, 1577-1578, 2007.10.2337/dc06-1998
  11. 11. Paisley K.E. et al. Endothelial dysfunction and inflammation in asymptomatic proteinuria. Kidney Int., 63, 624-633, 2003.10.1046/j.1523-1755.2003.00768.x
  12. 12. Ruggenenti P. et al. Reno-protective properties of ACE-inhibition in non-diabetic nephropathies with non-nephrotic proteinuria. Lancet, 354, 359-364, 1999.10.1016/S0140-6736(98)10363-X
  13. 13. Stehouwer C.D. et al. Microalbuminuria is associated with impaired brachial artery, flow-mediated vasodilation in elderly individuals without and with diabetes: further evidence for a link between microalbuminuria and endothelial dysfunction – the Hoorn Study. Kidney Int., 92, S42-S44, 2004.10.1111/j.1523-1755.2004.09211.x15485416
  14. 14. Webb N.J. et al. Randomized, double-blind, controlled study of losartan in children with proteinuria. Clin J Am Soc Nephrol., 5, 417-424, 2010.10.2215/CJN.06620909282756920089489
  15. 15. Zoccali C., Kielstein J.T. Asymetric dimethylarginine: A new player in the pathogenesis of renal disease? Curr Opin Nephrol Hypertens, 15, 314-320, 2006.10.1097/01.mnh.0000222701.22583.e816609301
DOI: https://doi.org/10.1515/cipms-2016-0038 | Journal eISSN: 2300-6676 | Journal ISSN: 2084-980X
Language: English
Page range: 180 - 183
Submitted on: Aug 11, 2016
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Accepted on: Oct 4, 2016
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Published on: Jan 23, 2017
Published by: Sciendo
In partnership with: Paradigm Publishing Services
Publication frequency: 4 issues per year

© 2017 Zbigniew Heleniak, Zbigniew Zdrojewski, Piotr Wisniewski, Leszek Bieniaszewski, Boleslaw Rutkowski, Alicja Debska-Slizien, published by Sciendo
This work is licensed under the Creative Commons Attribution-NonCommercial-NoDerivatives 3.0 License.