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Cardiovascular Implications of COVID-19 Infections Cover

Cardiovascular Implications of COVID-19 Infections

Open Access
|Apr 2020

Figures & Tables

Figure 1.

Representative chest computed tomography of a patient who was admitted with COVID-19.

Figure 2.

Schematic representing the cardiovascular manifestations of COVID-19. CA: coronary artery; VV ECMO: veno-venous extracorporeal membrane oxygenation; VA ECMO: veno-arterial ECMO; ACS: acute coronary syndrome; TTE: transesophageal echocardiogram; CTA: computed tomography angiography; TdP: Torsades de Pointes; RV: right ventricular

Figure 3.

Proposed mechanism of cardiovascular (CV) damage by SARS-CoV-2. ACE2: angiotensin-converting enzyme 2

Figure 4.

Common causes for elevated troponin level in patients with COVID-19. MI: myocardial infarction; ACS: acute coronary syndrome

Table 1.

The Tisdale score can be used to identify patients at high risk for developing drug induced QTc prolongation.31 K+: potassium; MI: myocardial infarction

RISK FACTORSPOINTS
Age ⩾ 68 years1
Female sex1
Loop diuretic1
Serum K+ ⩽ 3.5 mEq/L2
Admission QTc ⩾ 450 ms2
Acute MI2
⩾ 2 QTc-prolonging drugs3
Sepsis3
Heart failure3
One QTc-prolonging drug3
Maximum risk score21
Low risk: ⩽ 6 pts; moderate risk: 7–10 pts, high risk: ⩾ 11 pts
Language: English
Page range: 146 - 154
Published on: Apr 1, 2020
Published by: Houston Methodist DeBakey Heart & Vascular Center
In partnership with: Paradigm Publishing Services

© 2020 Akanksha N. Thakkar, Isaac Tea, Mouaz H. Al-Mallah, published by Houston Methodist DeBakey Heart & Vascular Center
This work is licensed under the Creative Commons Attribution-NonCommercial 4.0 License.