Practical method for determination of the U status of S–s– erythrocytes
Reid, M.E., Storry, J.R., Maurer, J., Nance, S.T.
Loss of enzyme-sensitive antigens due to the presence of leukocytes, neomycin sulfate, and LISS
Velliquette, R.W., Howard, P., Malyska, H., Reid, M.E.
The Cromer blood group system: a review
Storry, J.R., Reid, M.E., Yazer, M.H.
Molecular studies of DO alleles reveal that JO is more prevalent than HY in Brazil, whereas HY is more prevalent in New York
Castilho, L., Baleotti, W., Tossas, E., Hue-Roye, K., Ribeiro, K.R., Lomas-Francis, C., Charles-Pierre, D., Reid, M.E.
From DNA to blood groups
Reid, M.E.
RHD deletion in a patient with chronic myeloid leukemia
Murdock, A., Assip, D., Hue-Roye, K., Lomas-Francis, C., Hu, Z., Vege, S., Westhoff, C.M., Reid, M.E.
Molecular background of RH in Bastiaan, the RH:–31,–34 index case, and two novel RHD alleles
Reid, M.E., Hipsky, C.H., Velliquette, R.W., Lomas-Francis, C., Larimore, K., Olsen, C.
A review of the JR blood group system
Castilho, L., Reid, M.E.
Value of DNA-based assays for donor screening and regulatory issues
Strauss, D., Reid, M.E.
SC*994C>T causes the Scnull phenotype in Pacific Islanders and successful transfusion of Sc3+ blood to a patient with anti-Sc3
Hue-Roye, K., Velliquette, R.W., Larimore, K., Moscarelli, S., Ohswaldt, N., Lomas-Francis, C., Reid, M.E.
The polymorphism nt 76 in exon 2 of SC is more frequent in Whites than in Blacks
Fuchisawa, A., Lomas-Francis, C., Hue-Roye, K., Reid, M.E.
Consortium for Blood Group Genes (CBGG): 2009 report
Denomme, G.A., Westhoff, C.M, Castilho, L.M., St-Louis, M., Castro, V., Reid, M.E.